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Halotestin®


Androgenic 850
Anabolic 1900
ChemicalNames  
Estrogenic Activity none
Progestational Activity Activity no data available (low)
Presentation  

Description 
(10Mg/Tab Each Bottle contain 50 Tablets)

Fluoxymesterone is an oral anabolic steroid derived from testosterone. More specifically, it is a methyltestosterone derivative, differing by the addition of 11-beta-hydroxy and 9-alpha-fluoro groups. The result is a potent orally active non-aromatizable steroid that exhibits extremely strong ,androgenic properties. Fluoxymesterone is considerably more androgenic than testosterone, while at the same time the anabolic effects of this agent are considered to be moderate in comparison. This makes fluoxymesterone a great strength drug, but not the most ideal agent for gaining muscle mass. The predominant effects seen when taking fluoxymesterone are increased strength, increased muscle density, and increased definition, with only modest size increases.



Administration (Men): 

To treat androgen insufficiency, early prescribing guidelines for fluoxymesterone called for a dose of 2-10
mg per day. Modern prescribing guidelines call for a daily dosage of 5-20 mg. Therapy is usually initiated at the full 20 mg dosage, which is later adjusted downward to meet the individual needs of the patient. The drug would be continued long-term unless laboratory tests (lipids, liver enzymes, etc.) or side effects contraindicate its continued use. For physique- or performance-enhancing purposes, an effective oral daily dosage would fall in the range of 1040 mg, taken in cycles lasting no more than 6-8 weeks to minimize hepatotoxicity. This level is sufficient for measurable increases in muscle strength, which may be accompanied by modest increases in lean muscle mass. Fluoxymesterone is commonly used by athletes in weight-restricted sports like wrestling, powerlifting, and boxing, due to the fact that strength gained from the drug is usually not accompanied by great increases in bodyweight. When properly used, it can allow a
competitor to stay within a specified weight range, yet drastically improve his performance.  luoxymesterone is also commonly used for bodybuilding contest preparation. When the competitor has an acceptably low body fat percentage, the strong androgen level (in absence of excess estrogen) can elicit an extremely hard and defined ("ripped") look to the muscles. The shift in androgen/estrogen ratio additionally seems to bring about a state in which the body may be more inclined to burn off excess fat and prevent new fat storage. The "hardening" effect of fluoxymesterone would, therefore, be somewhat similar to that seen with trenbolone, although it will be without the same level of mass gain. In cutting phases, a milder anabolic such as DecaDurabolin ® or Equipoise® is commonly stacked with fluoxymesterone, as they provide good anabolic effect without excessive estrogen buildup. Here, fluoxymesterone provides a well-needed androgenic component, helping to promote a more solid and defined gain in muscle mass, with less interference with energy and libido, than might be obtained with a primarily anabolic agent alone. Perhaps Primobolan®-Depot would be an even better choice, as with such a combination there is no buildup of estrogen, and likewise even less worry of water and fat retention. For mass, one might alternately use an injectable testosterone. A mix of 400 mg per week of testosterone enanthate and 20-30 mg daily of fluoxymesterone, for example, often provides exceptional increases in strength and lean muscle mass. A more significant level of androgenic side effects usually accompanies such a combination, however, as both compounds exhibit strong androgenic activity in the body.

 

Administration (Women): 

Fluoxymesterone is most often used as a secondary medication during inoperable androgen-sensitive breast cancer, when other therapies have failed to produce a desirable effect.The dosage used for this application is 1040 mg per day. Virilizing effects are common at doses of only 10-15 mg per day in these patients. Fluoxymesterone is not recommended for women for physique- or performance-enhancing purposes due to its strong androgenic nature and tendency to produce virilizing side effects.



Clenbuterol is a widely used bronchodilator in many parts of the world. The drug is most often prepared in 20mcg tablets, but it is also available in syrup and injectable form. ClenbuteroClenbuterol is a widely used bronchodilator in many parts of the world. The drug is most often prepared in 20mcg tablets, but it is also available in syrup and injectable form. Clenbuterol belongs to a broad group of drugs knows as sympathomimetics. These drugs affect that sympathetic nervous system in a wide number of ways, largely mediated by the distribution of adrenoceptors. There are actually nine different types of these receptors in the body, which are classified as either alpha or beta and further subcategorized by type number. Depending on the specific affinities of these agents for the various receptors, they can potentially be used in the treatment of conditions such as asthma, hypertension, cardiovascular shock, arrhythmias, migraine headaches and anaphylactic shock. 

The drug clenburerol is specifically a selective bata-2 sympathomimetric , primarily affecting only one of the three subsets of beta-receptors. Of particular interest is the fact that this drug has little beta-1 stimulating activity. Since beta-1 receptors are closely tied to the cardiac effects of these agents, this allows Clenbuterol to reduce reversible airway obstruction (and effect of beta-2 stimulation) with much less cardiovascular side effect compared to non-selective beta agonists. Clinical studies with this drug show it is extremely effective as a bronchodilator, with a low level of user complaints and high patient compliance. Clenbuterol also exhibits an extremely long half-life in the body, which is measured to be approximately 34 hours long. This makes steady blood levels easy to achive, requiring only a single or twice daily dosing schedule almost. This of course makes it much easier for the patient to use, and may tie in to its high compliance rate. Despite that Clenbuterol is available in a wide number of other countries however; this compound has never been approved for use in the United States. The fact that there are a number o similar, effective asthma medications already available in this country may have something to do with this, as a prospective drug firm would likely not find it a profitable enough products to warrant undergoing the expense of the FDA approval process. Regardless, foreign Clenbuterol preparations are widely available on the U.S black market. 

In animal studies Clenbuterol is shown to exhibit anabolic activity, obviously an attractive trait to the athlete. This compound is additionally a known to directly stimulate fat cells and accelerate the breakdown of triglycerides to form free fatty acids. Its efficacy in this area makes Clenbuterol a very attractive, and today almost mandatory, pre-contest drug. Those interested in this drug are most often hoping it will impart a little of both benefits, promoting the loss of body fat while imparting strength and muscle mass increases. But as was well pointed out by a review published in the august 1995 issues of Medicine and Science in Sport and Exercise, the possible anabolic activities in humans are very questionable, and based only on animal data using much larger doses than would be required for bronchodilation. With such reports there has been a lot of debate lately as to whether or not Clenbuterol is really anabolic at all. Some seem to swear by the fact that it builds muscle regardless, firmly sticking by “clen” as a great off-season or adjunct anabolic. To others such reports are confirmation that athletes have wasted valuable time and money on drug that do not work as they are intended to by the user. 

This debate continues today, with many still using Clenbuterol as a potential anabolic. With this in mind athletes will tailor their dosage and cycling of this product individually depending on which of the two “possible” results are more desired, and how much side effects are to be tolerated. The possible side effects of Clenbuterol include those of other CNS stimulants, and include such occurrences as shaky hands, insomnia, sweating, increased blood pressure and nausea. These side effects will generally subside after a week or so of use however, once the user becomes accustomed to the drug. One would typically start a cycle by gradually increasing the dosage each day until a desired range is established. This process will minimize the unwanted side effects seen from the drug; which otherwise might be dramatic if a large dose is administered from the onset. Men generally end up in the range of 2-8 tablets per day, although some people do claim to tolerate even higher dosage. Women get by on less, generally 2-4 tablets daily. Very quickly, the drug will elevate the body temperature. The rise in not usually dramatic, perhaps a half of a degree or so, sometimes a little more. This elevation is due to your body burning excess energy (largely from fat) and is usually not uncomfortable. 

Now that it is working, the number of consecutive days Clenbuterol can be used is believed to be dependent on the goal of the individual. To be clean, the athletic benefits of this drug will only last for a limited time and then diminish, largely due to beta-receptor downregulation. When using it for fat loss, the primary effect of the drug, it seems to work well approximately 4-6 weeks. During this period, users will want to constantly monitor their body temperature elevation. Once the temperature drops back to normal, Clenbuterol is no longer exhibiting a thermogenic effect. At this point increasing the dosage would not very effective, and a break for at least a few weeks should be taken before it used again effectively. If one is looking for strength gains, Clenbuterol appears to be effective for a much shorter period of time, around 3-4 weeks. This may be due to an absence of real anabolic effect, with the strength gain seen with Clenbuterol possibly due only to the stimulant properties of the drug (similar to the strength boost seen by ephedrine users). 

Many competitors also find the fat burning effect of Clenbuterol can be further enhanced by additional substances. When combined with thyroid hormones, specifically the powerful Cytomel, the thermogenic effect can become extremely dramatic. This can be to a point that the athlete could shred exceptional amounts of extra fat during contest preparations, without a dramatic restriction in calories. Such a mix can be further used during a steroid cycle, eliciting a much harder look from the anabolics, These cutting agents can often greatly inhibit extra fat storage during the cycle, even when using strong aromatizing androgens. A Clenbuterol/thyroid mix is also common when using growth hormone, further enhancing the thermogenic and anabolic effect of this therapy. Ketotifen has also been an extremely popular adjunct to Clenbuterol therapy as of late which is an antihistamine that exhibits the peculiar and extremely welcome side effect of upregulating beta-2 receptor density. It seems capable of not only increasing the potency of each dose of Clenbuterol, but also preventing the rapid drop in thermogenic effectiveness that is attributed to receptor downregulation. l belongs to a broad group of drugs knows as sympathomimetics. These drugs affect that sympathetic nervous system in a wide number of ways, largely mediated by the distribution of adrenoceptors. There are actually nine different types of these receptors in the body, which are classified as either alpha or beta and further subcategorized by type number. Depending on the specific affinities of these agents for the various receptors, they can potentially be used in the treatment of conditions such as asthma, hypertension, cardiovascular shock, arrhythmias, migraine headaches and anaphylactic shock. 

The drug clenburerol is specifically a selective bata-2 sympathomimetric , primarily affecting only one of the three subsets of beta-receptors. Of particular interest is the fact that this drug has little beta-1 stimulating activity. Since beta-1 receptors are closely tied to the cardiac effects of these agents, this allows Clenbuterol to reduce reversible airway obstruction (and effect of beta-2 stimulation) with much less cardiovascular side effect compared to non-selective beta agonists. Clinical studies with this drug show it is extremely effective as a bronchodilator, with a low level of user complaints and high patient compliance. Clenbuterol also exhibits an extremely long half-life in the body, which is measured to be approximately 34 hours long. This makes steady blood levels easy to achive, requiring only a single or twice daily dosing schedule almost. This of course makes it much easier for the patient to use, and may tie in to its high compliance rate. Despite that Clenbuterol is available in a wide number of other countries however; this compound has never been approved for use in the United States. The fact that there are a number o similar, effective asthma medications already available in this country may have something to do with this, as a prospective drug firm would likely not find it a profitable enough products to warrant undergoing the expense of the FDA approval process. Regardless, foreign Clenbuterol preparations are widely available on the U.S black market. 

In animal studies Clenbuterol is shown to exhibit anabolic activity, obviously an attractive trait to the athlete. This compound is additionally a known to directly stimulate fat cells and accelerate the breakdown of triglycerides to form free fatty acids. Its efficacy in this area makes Clenbuterol a very attractive, and today almost mandatory, pre-contest drug. Those interested in this drug are most often hoping it will impart a little of both benefits, promoting the loss of body fat while imparting strength and muscle mass increases. But as was well pointed out by a review published in the august 1995 issues of Medicine and Science in Sport and Exercise, the possible anabolic activities in humans are very questionable, and based only on animal data using much larger doses than would be required for bronchodilation. With such reports there has been a lot of debate lately as to whether or not Clenbuterol is really anabolic at all. Some seem to swear by the fact that it builds muscle regardless, firmly sticking by “clen” as a great off-season or adjunct anabolic. To others such reports are confirmation that athletes have wasted valuable time and money on drug that do not work as they are intended to by the user. 

This debate continues today, with many still using Clenbuterol as a potential anabolic. With this in mind athletes will tailor their dosage and cycling of this product individually depending on which of the two “possible” results are more desired, and how much side effects are to be tolerated. The possible side effects of Clenbuterol include those of other CNS stimulants, and include such occurrences as shaky hands, insomnia, sweating, increased blood pressure and nausea. These side effects will generally subside after a week or so of use however, once the user becomes accustomed to the drug. One would typically start a cycle by gradually increasing the dosage each day until a desired range is established. This process will minimize the unwanted side effects seen from the drug; which otherwise might be dramatic if a large dose is administered from the onset. Men generally end up in the range of 2-8 tablets per day, although some people do claim to tolerate even higher dosage. Women get by on less, generally 2-4 tablets daily. Very quickly, the drug will elevate the body temperature. The rise in not usually dramatic, perhaps a half of a degree or so, sometimes a little more. This elevation is due to your body burning excess energy (largely from fat) and is usually not uncomfortable. 

Now that it is working, the number of consecutive days Clenbuterol can be used is believed to be dependent on the goal of the individual. To be clean, the athletic benefits of this drug will only last for a limited time and then diminish, largely due to beta-receptor downregulation. When using it for fat loss, the primary effect of the drug, it seems to work well approximately 4-6 weeks. During this period, users will want to constantly monitor their body temperature elevation. Once the temperature drops back to normal, Clenbuterol is no longer exhibiting a thermogenic effect. At this point increasing the dosage would not very effective, and a break for at least a few weeks should be taken before it used again effectively. If one is looking for strength gains, Clenbuterol appears to be effective for a much shorter period of time, around 3-4 weeks. This may be due to an absence of real anabolic effect, with the strength gain seen with Clenbuterol possibly due only to the stimulant properties of the drug (similar to the strength boost seen by ephedrine users). 

Many competitors also find the fat burning effect of Clenbuterol can be further enhanced by additional substances. When combined with thyroid hormones, specifically the powerful Cytomel, the thermogenic effect can become extremely dramatic. This can be to a point that the athlete could shred exceptional amounts of extra fat during contest preparations, without a dramatic restriction in calories. Such a mix can be further used during a steroid cycle, eliciting a much harder look from the anabolics, These cutting agents can often greatly inhibit extra fat storage during the cycle, even when using strong aromatizing androgens. A Clenbuterol/thyroid mix is also common when using growth hormone, further enhancing the thermogenic and anabolic effect of this therapy. Ketotifen has also been an extremely popular adjunct to Clenbuterol therapy as of late which is an antihistamine that exhibits the peculiar and extremely welcome side effect of upregulating beta-2 receptor density. It seems capable of not only increasing the potency of each dose of Clenbuterol, but also preventing the rapid drop in thermogenic effectiveness that is attributed to receptor downregulation.